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Fig. 1 | Biotechnology for Biofuels

Fig. 1

From: When metabolic prowess is too much of a good thing: how carbon catabolite repression and metabolic versatility impede production of esterified α,ω-diols in Pseudomonas putida KT2440

Fig. 1

a Mechanism of Crc/Hfq binding to attachment sites (AS) on mRNA of target genes. RBS: ribosome binding site. b Mechanism of catabolite repression on alkBGTL when glucose and n-alkanes are fed. AlkS activator of alkane degradation pathway, Crc global regulator of carbon catabolite repression, Cyo cytochrome o ubiquinol oxidase, B AlkB, alkane monooxygenase, F AlkF, rubredoxin which is indispensable for AlkB activity, G AlkG, rubredoxin, L AlkL, outer membrane protein, T AlkT, rubredoxin reductase, S AlkS, activator of alkane degradation pathway, PalkS2 promoter of AlkS with efficient transcription in presence of alkanes, PalkS1 promoter of AlkS in absence of alkanes, PalkB promoter of AlkB

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